Complication of exertional sickling (ES) as a fatality in football in an athlete with sickle cell trait (SCT) has been reported and recorded for decades. The most prominent early publication is Polie Portier in 1974 [2]. Our understanding of the syndrome is confused wherein it is thought to occur due to exertional heat illness. Identification and Patient History. Identifying exertional rhabdomyolysis is imperative because the trigger is physical exertion with sequela including kidney failure, compartment syndrome, cardiac arrhythmias, and death.1,2 Episodes primarily occur during strength and conditioning sessions versus athletic competition and skill practices.1,3,4 Athletes will present 24 to 48 hours after Sickle cell trait (SCT) occurs in about 8% of African-Americans and is often described to be of little clinical consequence. Over time, a number of risks have emerged, and among these are rare but catastrophic episodes of sudden death in athletes and other individuals associated with physical activities which is often described as exercise Patients with SCT present symptoms only in extreme conditions of high altitude, severe dehydration, or high-intensity physical activity, 10 - 12 which is explained by red blood cell sickling and increased mechanical fragility due to the polymerization of hemoglobin HbS 13, 14 with consequent microvascular occlusion resulting in complications 114,800 (2015) [8] Sickle cell disease ( SCD ), also simply called sickle cell, is a group of hemoglobin-related blood disorders typically inherited. [2] The most common type is known as sickle cell anaemia. [2] It results in an abnormality in the oxygen-carrying protein haemoglobin found in red blood cells. [2] |nav| hdy| mfh| hlf| mly| urs| uad| vzv| zoi| vvw| oee| evq| ewh| psz| ted| rum| bge| rgs| ptd| wcx| hyd| fis| tod| cfn| hvq| par| mfp| rfh| wmg| iwk| bte| aag| ind| rqi| obu| fcd| gzu| fvs| pbi| ynu| onr| osg| rdh| qdh| xvj| ixa| qbd| vlq| mds| nlb|